JNK signaling pathways is required for mitochondrial dysfunction-induced iinsulin resistance in WRL-68 human liver cell
Mitochondrial dysfunction and metabolic inflammation have emerged as the key events underpinning the development of obesity-related insulin resistance and type 2 diabetes. Accumulating evidence indicates that c-Jun NH(2)-terminal kinase (JNK) signaling pathways are among the regulatory pathways resp...
Saved in:
Main Authors: | Abu Bakar, Mohamad Hafizi, Sarmidi, Mohamad Roji, Yaakob, Harisun, Cheng, Kian Kai, Zaman Huri, Hasniza |
---|---|
Format: | Article |
Published: |
Mary Ann Liebert Inc
2015
|
Subjects: | |
Online Access: | http://eprints.utm.my/id/eprint/58458/ |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Amelioration of mitochondrial dysfunction-induced insulin resistance in differentiated 3T3-L1 adipocytes via inhibition of NF-κB pathways
by: Abu Bakar, Mohamad Hafizi, et al.
Published: (2014) -
Mitochondrial dysfunction as a central event for mechanisms underlying insulin resistance: the roles of long chain fatty acids
by: Abu Bakar, Mohamad Hafizi, et al.
Published: (2015) -
Heat sensitivity between human normal liver (WRL68)l(MCF7) cell lines
by: Hamdan, Salehhuddin, et al.
Published: (2013) -
Heat sensitivity between human normal liver (WRL68) and breast cancer (MDAMB 231) cell lines
by: Hamdan, Salehhuddin, et al.
Published: (2013) -
Heat sensitivity between human normal liver (WRL68) and breast cancer (MCF7) cell lines
by: Elengoe, A., et al.
Published: (2013)